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It means that you increase the number hypergenesis or hyperplasia, of cell / cell proliferation. The term can lead to total enlargement of the organ, which is mixed with benign tumors / benign tumors sometimes. It is the common hyperplasia’s become a stimulus. Microscopic cells are similar to normal cells, but the number will increase. Thereby increasing the size of the cells sometimes. Hyperplasia, hypertrophy is different in that the adaptive changes in cell hypertrophy associated whereas increasing hyperplasia, the number of cells, which increases the cell size. Hyperplasia, are considered to certain stimuli and physiological response (normal), cell over-growth is to remain subject to normal regulatory control mechanism. (The benign tumor processes and cancer-based) contrast to the tumor, which is propagated in a non-physiologically abnormal cells genetically does not respond to normal stimuli. As can be seen from the following examples, the physiological growth of cells, could be a pathological cause secondary in practice. However, the distribution itself is abnormal growth is very, in contrast, the tumor is a normal response of the abnormal state of the other.

Increasing demand (for example, to compensate for growth of the basal layer of the epidermis, the loss of the skin), hyperplasia or other, due to include chronic inflammatory reaction, dysfunction hormones, and any number of reasons it is possible to compensate for damage or disease site. there is a possibility that hyperplasia is a harmless, occur in a particular organization. Preparation of future nursing, example of a successful response is playing an over-growth of mammary secretory cells in the chest in response to pregnancy by it.

Further hyperplasia, it can be artificially induced by hormones, and injecting the human growth hormone, such as IGF-1. IGF strong effect is also the ability to cause hyperplasia, which is a division of the actual cell of the human body on the most interesting and perhaps. On the other hand, the use of steroids and weight training, in skeletal muscle cells to increase the size of the cell simply, for example, what is to happen to hypertrophy. Using the IGF, then one, it is possible to induce hyperplasia increases the number of muscle cells present in the tissue. Weight training, you can mature in size and strength to new these cells with or without the use of anabolic steroids. In addition, animal studies, can cause hyperplasia of this phenomenon has not been confirmed in humans still stretching the muscles are shown. Increases the number of muscle fibers, thereby not to increase the size of the single fiber, hyperplasia may be induced by specific training capacity of athletic performance.

Endometrial hyperplasia is a condition of excessive growth of cells in the lining or inner membrane of the uterus. Most cases of endometrial hyperplasia due to high levels of counter-proliferation effect usually, a combination of estrogen and a sufficiently high level of hormones such as progesterone, estrogen on the fabric. This (e.g., granulosa cell tumors) can be carried out in a number of conditions including the particular form of estrogen replacement therapy and obesity, polycystic ovary syndrome, estrogen-producing tumor. Endometrial hyperplasia is also a co-existence of endometrial cancer or factors, for serious risk development, treatment and observation since it is carefully essential that women with this condition.

Woman Baby classical CAH was blurred genital exposure due to high concentration of intrauterine androgen. 21 – CAH by hydroxylase deficiency is the most common cause of ambiguous genitalia genotype in normal girls (46XX). In response to less severely affected, may present with early pubarche women. I can young women with symptoms (menstruation, polycystic ovaries, hirsutism) of polycystic ovary syndrome. Usually, there is no indication of CAH at birth male classical CAH. Some may present a possible extension of the penis and pigmentation. Old man with a diagnosis of CAH is different, I will depend on the severity of aldosterone deficiency. Boys announcement and salt consumption at the beginning of the symptoms of hypovolemia and hyponatremia,. Boy presented later with signs of virilization When the non-salt wasting disease.

Cortisol is an adrenal steroid hormones necessary for normal endocrine function. Production begins in the second month of fetal life. Bad cortisol is characteristic of most forms of CAH. I increase the ACTH as a result of inefficiencies in cortisol levels can lead to hyperactivity proliferation of steroid-producing cells of the adrenal cortex (hyperplasia) in order. There is a defect that causes congenital adrenal hyperplasia.

Steroid. Enzymes involved in CAH is represented by a bar of 4 green and red in the upper half of the scheme. Will be reduced by the mineralocorticoid or androgen production (bottom left) by the possible enzyme. By this, it can lead to increased production of other molecules due to the accumulation of the precursor. Usually, lack of cortisol in CAH is not a problem with the unit minutes most serious those involved. Androgen as testosterone and estrogen, such as estradiol and synthetic process, synthesis of cortisol shares of Mine, such as aldosterone like this. Or subject to excessive deficit, production of three classes of these hormones cause the most important issues for people with CAH. Specific enzyme is inefficient associated with certain patterns lack of sex steroids and mineralocorticoids or higher.